Harrison M, Odell EW, Agrawal M Gingival fibromatosis with prune-belly syndrome. Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 1998; 86:304-307 https://doi.org/10.1016/s1079-2104(98)90176-7
Beaumont J, Chesterman J, Kellett M, Durey K. Gingival overgrowth: Part 1: aetiology and clinical diagnosis. Br Dent J. 2017; 222:85-91 https://doi.org/10.1038/sj.bdj.2017.71
Ghartimagar D, Koirala U, Ghosh A Squamous cell carcinoma arising in familial gingival fibromatosis-a rare case report. J Surg Case Rep. 2017; 2017 https://doi.org/10.1093/jscr/rjx197
Doufexi A, Mina M, Ioannidou E. Gingival overgrowth in children: epidemiology, pathogenesis, and complications. A literature review. J Periodontol. 2005; 76:3-10 https://doi.org/10.1902/jop.2005.76.1.3
Ferreira Gonçalves C, Mundim AP, Martins RFS Hereditary gingival fibromatosis: a case report with seven-year follow-up. Acta Stomatol Croat. 2018; 52:254-258 https://doi.org/10.15644/asc52/3/9
Hereditary gingival fibromatosis (HGF) is a benign, non-haemorrhagic, fibrous gingival overgrowth of unknown cause. Clinically, gingivae can be seen to partially or fully cover teeth, and can limit oral function, for example eating, speaking, and maintaining oral hygiene. HGF normally starts in the transition between the primary and secondary dentition. Surgical excision is often required. This case describes the management of a 37-year-old male with generalized HGF with extensive bilateral palatal growths extending into the oropharynx; not a reported presentation of HGF. Awareness of the condition and familial history could aid management at an earlier stage.
CPD/Clinical Relevance: This article describes the management of an uncommon presentation of hereditary gingival fibromatosis in the palate that affected the patient's oral function.
Article
Hereditary gingival fibromatosis (HGF), also known as hereditary gingival hyperplasia, is a benign, non-haemorrhagic gingival overgrowth. It can make the maxilla and mandible appear bulky, fully or partially cover teeth, and alter the function of the oral cavity.1,2 Its prevalence is relatively low (1:175,000) and the condition can present with or without syndromic associations.1,3 There is no evidence that it affects the alveolus; however, when tissues become plaque retentive, a secondary periodontal disease can occur.
HGF normally appears with the eruption of the permanent dentition.1 Classically, it presents with no inflammation, a fibrous appearance, and marked stippling. HGF can be generalized or localized to specific sites, typically the maxillary tuberosities or the labial gingivae around the mandibular molars.1
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