References

Treatment of plaque-induced gingivitis, chronic periodontitis, and other clinical conditions. J Periodontol. 2001; 72:1790-1800
British Society of Periodontology. http://www.bsperio.org.uk/gpg/story_html5.html (Accessed April 2015)
Armitage GC. The complete periodontal examination. Periodontology 2000. 2004; 34:22-33
Axelsson P, Nyström B, Lindhe J. The long-term effect of a plaque control program on tooth mortality, caries and periodontal disease in adults. Results after 30 years of maintenance. J Clin Periodontol. 2004; 31:749-757
Wilson TG, Glover ME, Schoen J, Baus C, Jacobs T. Compliance with maintenance therapy in a private periodontal practice. J Periodontol. 1984; 55:468-473
Preshaw PM, Heasman L, Stacey F, Steen N, McCracken GI, Heasman PA. The effect of quitting smoking on chronic periodontitis. J Clin Periodontol. 2005; 32:869-879
Heasman L, Stacey F, Preshaw PM, McCracken GI, Hepburn S, Heasman PA. The effect of smoking on periodontal treatment response: a review of clinical evidence. J Clin Periodontol. 2006; 33:241-253
Walker CB. The acquisition of antibiotic resistance in the periodontal microflora. Periodontology 2000. 1996; 10:79-88
Teughels W, Dhondt R, Dekeyser C, Quirynen M. Treatment of aggressive periodontitis. Periodontology 2000. 2014; 65:107-133
Herrera D, Alonso B, León R, Roldán S, Sanz M. Antimicrobial therapy in periodontitis: the use of systemic antimicrobials against the subgingival biofilm. J Clin Periodontol. 2008; 35:45-66
Griffiths GS, Ayob R, Guerrero A, Nibali L, Suvan J, Moles DR, Tonetti MS. Amoxicillin and metronidazole as an adjunctive treatment in generalized aggressive periodontitis at initial therapy or re-treatment: a randomized controlled clinical trial. J Clin Periodontol. 2011; 38:43-49
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Diagnosis and management of chronic and aggressive periodontitis part 2: periodontal management

From Volume 44, Issue 5, May 2017 | Pages 402-408

Authors

Despoina Chatzistavrianou

DDS MFDS RCSEd, MClinDent Pro, MPros RCSEd

Specialist in Prosthodontics, Specialty Registrar in Restorative Dentistry, Birmingham Dental Hospital and University of Birmingham School of Dentistry, Birmingham Community Healthcare NHS Trust, Birmingham, UK

Articles by Despoina Chatzistavrianou

Fiona Blair

BDS, LDS, FDS(Rest) RCPS, MSc, DRD, MRD

Consultant in Restorative Dentistry, Birmingham Dental Hospital, St Chad's Queensway, Birmingham B4 6NN, UK

Articles by Fiona Blair

Abstract

The first paper of this three-part series discussed periodontal disease pathogenesis and highlighted elements in the clinical assessment which will help the clinician to establish the diagnosis of chronic and aggressive periodontitis. This second paper will focus on the management of chronic and aggressive periodontitis. Finally, the diagnosis and management of chronic and aggressive periodontitis will be reviewed in the third part of the series using two clinical examples.

CPD/Clinical Relevance: This paper aims to provide the general dental practitioner with an understanding of the aim of periodontal treatment, the management of chronic and aggressive periodontitis and the prognosis of periodontally involved teeth.

Article

The aim of periodontal treatment is to maintain the remaining periodontal tissues and improve gingival health.1 The treatment strategy for managing periodontal disease includes:

This is primarily achieved through non-surgical treatment. A further corrective phase may include periodontal surgery and finally the successfully treated patient enters the maintenance phase.1

Following the clinical assessment and the establishment of the diagnosis of periodontitis, periodontal management should involve delivery of a phased treatment with steps as follows:

An effective maintenance programme should include:

There are a number of factors that affect prognosis, with position in arch affecting likelihood of tooth loss, risk being greatest for maxillary second molars and least for mandibular canines (Table 1).23,24 Teeth with increased probing depth, mobility, furcation involvement, unsatisfactory crown-to-root ratio, malposition and those used as fixed abutments have worse initial prognosis.25 Prognosis can change over time, with good oral hygiene being a critical positive factor, and mobility decreasing likelihood for improvement. Continued smoking doubles the likelihood of a worsening prognosis.25 During maintenance, phase sites with bleeding on probing have three times higher risk of attachment loss compared to non-bleeding sites.26 Documented monitoring with indices is required to alert to early intervention, as presence of plaque deposits and bleeding on probing adversely affects prognosis, with 16% or more BOP sites reported as likely to lose further clinical attachment.26

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