References

Cancer Research UK [Internet]. 2015.
Cancer Research UK [Internet]. 2013.
Mehanna H, Jones T, Gregoire V, Ang KK Oropharyngeal carcinoma related to human papillomavirus. Br Med J. 2010; 340:(10)
Nelson BS, Heischober B Betel nut: a drug used by naturalized citizens from India, Far East Asia, and the South Pacific Islands. Ann Emerg Med. 1999; 34:(2)238-243
Marshall M An overview of drugs in oceania. In: Lindstrom L Lanham: University Press of America; 1987
Ekanayaka RP, Tilakaratne WM Oral submucous fibrosis: review on mechanisms of pathogenesis and malignant transformation. J Carcinogene Mutagene. 2013; S5
Lewis MAO, Jordan RCK, 2nd edn. London: BDA; 2012
Chu NS Effects of Betel chewing on the central and autonomic nervous systems. J Biomed Sci. 2001; 8:(3)229-236
Hsu HF, Tsou TC, Chao HR, Shy CG, Kuo YT, Tsai FY Effects of arecoline on adipogenesis, lipolysis, and glucose uptake of adipocytes – a possible role of betel-quid chewing in metabolic syndrome. Toxicol Appl Pharm. 2010; 245:(3)370-377
Taylor RF, Al-Jarad N, John LM, Conroy DM, Barnes NC Betel-nut chewing and asthma. Lancet. 1992; 339:(8802)1134-1136
Senn M, Baiwog F, Winmai J, Mueller I, Rogerson S, Senn N Betel nut chewing during pregnancy, Madang province, Papua New Guinea. Drug Alcohol Depen. 2009; 105:(1–2)126-131
Akhtar S Areca nut chewing and esophageal squamous-cell carcinoma risk in Asians: a meta-analysis of case-control studies. Cancer Cause Control. 2013; 24:(2)257-265
Her Majesty's Government [Internet]. 2014.
Brennan A, Meng Y, Holmes J, Hill-McManus D, Meier PS Potential benefits of minimum unit pricing for alcohol versus a ban on below cost selling in England 2014: modelling study. Br Med J. 2014; 349:(1)1-14
Croucher R, Awojobi O, Dahiya MLondon: Barts and the London School of Medicine and Dentistry; 2009
London: The Stationery Office; 2009
Panesar SS, Gatrad R, Sheikh A Smokeless tobacco use by South Asian youth in the UK. Lancet. 2008; 372:(9633)97-98
Eaton DK, Kann L, Kinchen S, Shaklin S, Ross J, Hawkins J Youth risk behavior surveillance – United States, 2009. MMWR Surveill Summ. 2010; 59:(5)1-142
Directive 2001/37/EC of the European Parliament and of the Council of 5 June 2001 on the approximation of the laws, regulations and administrative provisions of the Member States concerning the manufacture, presentation and sale of tobacco products – Commission statement. OJ L 194. 2001;
2014.
Ebbert J, Montori VM, Erwin PJ, Stead LF Interventions for smokeless tobacco use cessation. Cochrane Database Syst Rev. 2011; 16:(2)1-51
National Institute of Health and Care Excellence [Internet]. 2012.

Tackling the use of supari (areca nut) and smokeless tobacco products in the south asian community in the united kingdom

From Volume 43, Issue 5, June 2016 | Pages 442-447

Authors

Milan Chande

BDS

Dental Foundation Trainee, Boutique Dental 23, 23 Bryn Street, Ashton-in-Makerfield, Wigan, WN4 9AX, UK

Articles by Milan Chande

Krishna Suba

Fourth Year Dental Student, School of Dentistry, University of Manchester, Oxford Road, Manchester, M13 9PL, UK

Articles by Krishna Suba

Abstract

The use of supari (areca nut) and smokeless tobacco products are seen as a major risk factor for oral cancer. There are increasing rates of oral cancer across the United Kingdom, along with the increase of the use of these products. This article examines the uses of such products amongst the South Asian Community and explores sensitive issues associated with the cessation of their use. Evidence-based recommendations are provided on how to provide advice and treatment to patients that regularly use these products. A rethink is also suggested on the policy of taxation of such products.

CPD/Clinical Relevance: With the rates of oral cancer increasing across the United Kingdom, it is important for us as dental professionals to tackle the use of areca nut and smokeless tobacco products.

Article

A diagnosis of oral cancer can have a significant impact upon a person's life. Ensuring that patients have the best possible advice available to them, in order to reduce the risk of developing oral cancer, should be fundamental in practice. Whilst smoking is a major factor in developing oral cancer and lung cancer, recent evidence has shown that the incidence of lung cancer has begun to decrease, since more people have ceased smoking and fewer are taking up this dangerous habit.1 It therefore may seem unusual that there is an increase in the incidence of oral cancer.2 There are two recently identified risk factors that are contributing towards this increase.

First, human papillomavirus-linked oropharyngeal cancer is on the increase.3 Another factor, influenced by the increasing migrant population in the United Kingdom, is the availability of carcinogenic products: smokeless tobacco and areca nut, also known as ‘betel nut’ or ‘supari’.

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